Face and jaw emergencies
Facial cellulitis
- Di/flucloxacillin or Cephalexin
- Penicillin allergy: Clindamycin
- Suspected MRSA: Bactrim, clindamycin, doxycycline
- Penicillin allergy: Clindamycin
Erysipelas
- 70% lower extremities
- Classically described as disease of the face
- Nasopharynx is the typical source of bacteria
- Mostly S. pyogenes
- More severe form due to MRSA (bullous)
- Rx: Amoxicillin
Impetigo
- Discrete, superficial bacterial epidermal infection characterised by amber crusts or bullae
- Mostly in children
- S. aureus +- S. pyogenes (Group A beta-haemolytic)
- Bullous always staph aureus
- If uncomplicated, topical muporicin recommended
- If extensive or bullous – Flucloxacillin
- MRSA: Bactrim or clindamycin
Salivary gland infection
- Anatomy
- Parotid, submandibular and sublingual
- Facial nerve passes through superficial portion of parotid gland
- Stensen’s duct (from parotid) opens into mouth opposite second upper molar
- Submandibular and sublingual glands lie below tongue
- Submandibular ducts open into mouth either side of frenulum
- Multiple sublingual ducts open into the sublingual fold or directly into the submandibular duct
- Signs of infections
- Unilateral or bilateral facial swelling
- Multiple gland involvement suggests infection
- Palpable tender mass may be a sign of tumor or stone
- Dry mouth and eyes, recurrent episodes or joint symptoms suggest underlying Sjogren’s/collagen vascular disorders or immunological conditions
Salivary gland
- DDx of salivary gland swelling
- Infectious
- Viral parotitis (mumps) – Unilateral tense swelling; absent warmth
- Buccal cellulitis – Hib in non-immunised. Erythematous/tender
- Suppurative parotitis – Fever, pain, swelling, pus expression from Stensen’s duct
- Masseter space abscess: Dental infection with trismus, posterior inferior facial swelling
- TB – Chronic crusting plaques
- Immunologic
- Sjogren’s – Dry eyes/mouth/sclerosis
- Systemic lupus
- Sarcoidosis
- Neoplasm – No warm/erythema
- Sialolithiasis – Dehydration, chronic illness, swelling, tenderness, no warmth/fluctuance
- Infectious
Viral parotitis (mumps)
- Unilateral or bilateral parotid swelling
- Mostly paramyxovirus or less commonly influenzae, parainfluenza, coxsackie, echovirus, HIV
- Most common under 15yo
- For mumps (paramyxovirus)
- Prodromal fever, malaise, headache, myalgias, arthralgias, anorexia for 3-5 days
- The salivary gland swelling with tense and painful parotid (but no erythema/warmth)
- Stensen’s duct may be inflamed but no pus expressed
- Resolves over 1-5 days
- Contagious for 9 days from onset of parotid swelling
- Unilateral orchitis can occur in 20-30% of adult males
- Oophoritis in only 5% of females
Suppurative parotitis
- Serious bacterial infection in salivary gland with compromised salivary flow
- Caused by retrograde migration of oral bacteria into salivary ducts and parenchyma
- Risk factors
- Recent anaesthesia, dehydration, advanced age
- Sialolithiasis, oral neoplasms, salivary duct strictures, tracheostomy and ductal foreign bodies
- Medications that cause dehydration or decreased salivary flow
- Diuretics, antihistamines, TCA’s, phenothiazines, beta-blockers and barbiturates
- Chronic illnesses
- Renal failure, HIV, hepatic failure, DM, hypothyroidism, malnutrition, Sjogren’s, depression, anorexia, bulimia, hyperuricaemia and CF
Suppurative parotitis
- Mostly S. aureus +- S. pneumoniae, S. pyogenes, H. influenzae and anerobes (Bacteroides, Peptostreptococcus, fusobacteria) in 43%
- In immunocompromised, E. coli and Pseudomonas are common
- Red, tender parotid gland with rapid onset
- Pus from Stensen’s
- Often fever and trismus
- US and CT can rule out abscess but otherwise unhelpful
Suppurative parotitis
- Treatment aims at optimising salivary flow with hydration, massage and heat application to gland
- Lemon drops
- Discontinue drugs that cause dry mouth
- Oral antibiotics if not systemically unwell
- IV antibiotics if trismus, cannot tolerate oral liquids, immunocompromised or failed outpatient therapy
- Rx – Augmentin or Cephalexin + Metronidazole
- Add vancomycin if risk of MRSA
Sialolithiasis
- Calcium carbonate or calcium phosphate sialoliths in stagnant duct
- Mostly men in 3rd to 6th decades
- 80% occur in submandibular gland with rest in parotid
- Difficult to differentiate this from suppurative parotitis
- Colicky pain exacerbated by meals (salivation)
- Stone may be palpated in duct and gland feels firm
- Intra-oral radiographs are more sensitive than extra-oral films for visualising stone
- Calculi are radio-opaque in 70%
- US and thin-cut CT may also identify sialoliths but usually only done to rule out abscess formation
- Treatment
- Outpatient analgesia, antibiotics if suspicious for infection, lemon drops, digital milking of palpable stones
Masticator space infection
- Four potential spaces surrounded by muscles of mastication
- Masseteric, superficial temporal, deep temporal and pterygomandibular spaces
- All contiguous
- Bacteria may gain entry from dental infections, trauma, surgery or injections
- Polymicrobial and often anaerobic
- Strep, peptostreptococcus, Bacteroides, Prevotella, Fusobacterium, Actinomyces
Masticator space infection
- Presentation
- Acute unilateral facial swelling, pain, erythema and trismus
- Masster space infection
- Swelling appears posteroinferiorly on face with mild to moderate trismus
- Temporal space infection
- Soft tissue swelling of temporalis muscle is seen
- Trismus without swelling suggests pterygomandibular space infection
- May have constitutional signs or sepsis
Masticator space infection
- USS
- Can help differentiate abscess from cellulitis and to identify internal jugular thrombosis
- Contrast CT is prefered to rule out deep space infection (not sensitive for retropharyngeal space however)
- MRI can be considered if infection thought to reach the skull base
- Treatment
- Tissue planes extend down to neck/mediastinum so need to identify extent of infection early and begin treatment promptly (depending on clinical presentation)
- IV antibiotics – Clindamycin 600mg TDS
- PO Augmentin
Temporomandibular joint disorders
- Synovial joint with meniscus and hinge/glide action
- TMJ dysfunction
- Pain of joint and surrounding anatomical structures
- Degenerative joint disease can occur with chronic internal derangement or RA/SLE
- Chief complaint is usually pain with chewing
- Masseter muscle is most frequently identified painful area, then temporalis, SCM, splenius capitis and trapezius
- May have limited mandibular movement on examination
- DDx: Dental infection/trauma, jaw fracture/dislocation, otologic referred pain, temporal arteritis
- OPG is good first line imaging
- CT is useful for assessment of possible neoplasm, complex fractures or infections
- Simple analgesics are first-line if no evidence of infection/fracture with GP follow-up +- maxillofacial referral for definitive care
Trigeminal neuralgia
- Unknown cause
- Paroxysms of severe unilateral pain lasting only seconds with normal neurological examination
- No pain between paroxysms
- Classic
- Idiopathic cases and those due to microvascular compression
- Secondary
- Due to tumor, MS or other structural abnormalities
- History is key as pain is shocking, stabbing, brief, lasting seconds to minutes
- Triggers include light touching, chewing or light breeze
- Treatment
- Carbamzapine 100mg BD increased as required is useful
- Baclofen also proven successful
- Refer to neurology
Bell’s palsy
- Acute unilateral upper and lower facial palsy, posterior auricular pain, decreased tearing, hyperacusis and otalgia
- Can develop some symptoms prior to paralysis
- Peak of symptoms within 48 hours
- Careful neuro exam to rule out stroke, examine ear for Ramsay-Hunt
- Stroke, GBS and Ramsay-Hunt constitute 85% of misdiagnoses
- Treatment
- Prednisone 1mg/kg for 7 days then 10-day taper if within 72 hours of onset as improves chance of recovery
- Antivirals may be useful with steroids but not proven
- Ocular lubricants, tape patients eyelid in sleep and ophthal follow-up
Mandible dislocation
- May be anterior, posterior, lateral or superior
- Anterior dislocation is most common when mandibular condyle is forced in front of articular eminence
- Muscular spasm then traps mandible in this position
- Risk factors include shallow glenoid fossa, seizure and loss of joint capsule tone from previous trauma
- Usually bilateral
- Posterior dislocations are rare with condylar head in external auditory canal
- Lateral dislocations often associated with fracture, with condylar head forced laterally and then superiorly into temporal space
- Superior dislocations forces condylar head up into temporal space
Mandible dislocation
- If dislocation is unilateral, there will be deviation of jaw away from dislocation
- If posterior dislocation considered, examine the external auditory canal
- Posterior, lateral and superior dislocations = severe trauma
- If cooperative, atraumatic spontaneous anterior dislocation, diagnosis is clinical
- In all others, get OPG +- CT
- Perform reduction in ED for closed anterior dislocations without fracture
- Short-acting IV muscle relaxant e.g. midazolam may assist with muscle spasm
- LA using 21G needle into preauricular depression just anterior to tragus 2mL 2% lignocaine may obviate need for procedural sedation
Reduction of anterior TMJ dislocation
- Patient seated with head firmly against wall or chair back
- Apply gauze over gloved thumbs in case mandible snaps shut after relocation
- Facing patient, place thumbs over occlusal surface of mandibular molars as far back as possible
- Apply pressure down and back
- Slight opening of jaw may help disengage condyle from anterior eminence
- If bilateral, relocate one side at a time
- If successful, should be able to close mouth immediately. Post-reduction radiographs not mandatory
- D/C home, soft diet, caution in opening mouth >2cm for 2 weeks
- Support mandible with hand when yawning
- Elective referral to maxfax is recommended
Last Updated on October 6, 2020 by Andrew Crofton
Andrew Crofton
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